April 2nd, 2005

umm, hewwo?!

Chest pain correlating with ED?

I don't know if this is the proper place to ask this, but I have a question about chest pains, and any correlations they may have with an eating disorder.

I woke up this morning around six, and my legs were very crampy and I also noticed my chest hurt. I texted the girl I was going to go work out with to tell her I wouldn't be making it, and fell asleep shortly thereafter. I woke up again around seven, after my mom played a clever April Fool's joke on me. Irrelevant, sorry. Anyhow, I was trying to think of reasons to stay home because my body was dragging and I was so very tired, and I noticed my chest still hurt. I ended up sleeping until eleven, and then going to school.

The pain subsided throughout the day, but while I was at work tonight, it came back. I don't know how to describe the pain, it is kind of a dull ache. It's about 12:30 right now and my chest still hurts(and sleep seems to evade me), so I thought I'd come and ask if there is any correlation to my eating disorder.

Also, do your muscles cramp because of a potassium deficiency? Or why is this? I've never known my legs to cramp randomly when I'm not doing any strenuous activity, unless I get a "charlie horse".

Thank you.

(x posted to ed_medfacts)
house

(no subject)

So I was on handbag.com checking my BMI (that's all I was doing, I swear! I don't ever waste time on daft quizzes or celebrity hairstyles!) and I came across Collapse )

I just thought I'd share, because I recall a discussion from a while ago talking about changing regulations in clothing sizes. Thought this'd tie in nicely - and since it's not a medical type thing you can feel free to switch your brain off and join me at my level ;)
Hope y'all find it semi-interesting.
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eating disorders as addictions?

Excerpt from:
Addiction and the Eating Disorders
by Caroline Davis, Ph.D.
Psychiatric Times February 2001 Vol. XVIII Issue 2


Others have suggested that the eating disorders are, themselves, a form of drug addiction since their characteristics satisfy all the clinical and biological criteria for conventional addictions such as smoking, alcoholism and cocaine abuse (Davis and Claridge, 1998; Davis et al., 1999; Marrazzi and Luby, 1986). Foremost among these is the progressively compulsive nature of the behavior, even in the face of adverse consequences to health and safety (Heyman, 1996; Robinson and Berridge, 1993). Moreover, with continual exposure, individuals typically require more of the behavior to produce the same reinforcing effect (Berridge and Robinson, 1995). They also tend to experience an obsessively increasing craving for the behavior that can persist even after a long period of abstinence. Presumably that accounts, at least in part, for the fact that addicts have a strong tendency to resume the addictive behavior after treatment and for the chronic relapsing nature of addiction (Robinson and Berridge, 1993). These characteristics find direct parallels in the core eating-disorder behaviors such as dieting, over-exercising and binge eating, all of which tend to become increasingly excessive over time. Patients also report a strong compulsion to continue these behaviors despite serious medical complications, which is reflected in their prolonged morbidity and the high rate of relapse (Herzog et al., 1999; Strober et al., 1999).

At the biological level, similarities are also evident. We know, for instance, that strenuous exercise and starvation activate the dopaminergic (DA) reward pathway of the brain (Bergh and Sodersten, 1996; Casper, 1998). The resulting biological events underlie the auto-addiction opioid theory, which proposes that a chronic eating disorder is an addiction to the body's production of endogenous opioids and therefore is identical to the physiology and psychology of substance abuse in general (Huebner, 1993; Marrazzi and Luby, 1986). In other words, starving, bingeing and exercise all serve as drug delivery devices since they increase circulating levels of ß-endorphins that are chemically identical to exogenous opiates, and these endorphins are as potentially addictive because of their ability to stimulate DA in the brain's mesolimbic reward centers.

Via a different route, self-starving may have other biologically rewarding properties, albeit as a negative reinforcer. For example, in certain individuals, food restriction is reported to reduce anxiety. It has been suggested this might occur because of reduced serotonin activity in those with overactivity in this neurotransmitter system (Kaye, 1999).


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